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Results 1-10 of 21 (Search time: 0.004 seconds).
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PreviewIssue DateTitleAuthor(s)
2001Regulation of calcitonin receptor by glucocorticoid in human osteoclast-like cells prepared in vitro using receptor activator of nuclear factor-kB ligand and macrophage colony-stimulating factorWada, S.; Yasuda, S.; Nagai, T.; Maeda, T.; Kitahama, S.; Suda, S.; Findlay, D.; Iitaka, M.; Katayama, S.
1997Regulation by calcitonin and glucocorticoids of calcitonin receptor gene expression in mouse osteoclastsWada, S.; Udagawa, N.; Akatsu, T.; Nagata, N.; Martin, T.; Findlay, D.
2005Human trabecular bone-derived osteoblasts support human osteoclast formation in vitro in a defined, serum-free mediumAtkins, G.; Kostakis, P.; Welldon, K.; Vincent, C.; Findlay, D.; Zannettino, A.
2001The osteoclastogenic molecules RANKL and RANK are associated with periprosthetic osteolysisHaynes, D.; Crotti, T.; Potter, A.; Loric, M.; Atkins, G.; Howie, D.; Findlay, D.
2005Osteoprotegerin (OPG) is localized to the Weibel-Palade bodies of human vascular endothelial cells and is physically associated with von Willebrand factorZannettino, A.; Holding, C.; Diamond, P.; Atkins, G.; Kostakis, P.; Farrugia, A.; Gamble, J.; To, L.; Findlay, D.; Haynes, D.
1996Physiological Levels of Calcitonin Regulate the Mouse Osteoclast Calcitonin Receptor by a Protein Kinase A-Mediated MechanismWada, S.; Udagawa, N.; Nagata, N.; Martin, T.; Findlay, D.
1996Calcitonin Receptor Down-Regulation Relates to Calcitonin Resistance in Mature Mouse OsteoclastsWada, S.; Udagawa, N.; Nagata, N.; Martin, T.; Findlay, D.
1996Homologous Regulation of the Rat C1a Calcitonin Receptor (CTR) in Nonosteoclastic Cells is Independent of CTR Messenger Ribonucleic Acid Changes and Cyclis Adenosine 3', 5' -Monophospate-Dependent Protein Kinase ActivationFindlay, D.; Houssami, S.; Christopoulos, G.; Sexton, P.
2004CalcitoninFindlay, D.; Sexton, P.
1997Calcitonin receptors, bone sialoprotein and osteopontin are expressed in primary breast cancersGillespie, M.; Thomas, R.; Pu, Z.Y.; Zhou, H.; Findlay, D.

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