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PreviewIssue DateTitleAuthor(s)
2001Osteoprotegerin and receptor activator of nuclear factor kappaB ligand (RANKL) regulate osteoclast formation by cells in the human rheumatoid arthritic jointHaynes, D.; Crotti, T.; Loric, M.; Bain, G.; Atkins, G.; Findlay, D.
2001Regulation of calcitonin receptor by glucocorticoid in human osteoclast-like cells prepared in vitro using receptor activator of nuclear factor-kB ligand and macrophage colony-stimulating factorWada, S.; Yasuda, S.; Nagai, T.; Maeda, T.; Kitahama, S.; Suda, S.; Findlay, D.; Iitaka, M.; Katayama, S.
1997Regulation by calcitonin and glucocorticoids of calcitonin receptor gene expression in mouse osteoclastsWada, S.; Udagawa, N.; Akatsu, T.; Nagata, N.; Martin, T.; Findlay, D.
2005Human trabecular bone-derived osteoblasts support human osteoclast formation in vitro in a defined, serum-free mediumAtkins, G.; Kostakis, P.; Welldon, K.; Vincent, C.; Findlay, D.; Zannettino, A.
2001The osteoclastogenic molecules RANKL and RANK are associated with periprosthetic osteolysisHaynes, D.; Crotti, T.; Potter, A.; Loric, M.; Atkins, G.; Howie, D.; Findlay, D.
1999Bidirectional signaling between stromal and hemopoietic cells regulates interleukin-1 expression during human osteoclast formationHaynes, D.; Atkins, G.; Loric, M.; Crotti, T.; Geary, S.; Findlay, D.
1996Physiological Levels of Calcitonin Regulate the Mouse Osteoclast Calcitonin Receptor by a Protein Kinase A-Mediated MechanismWada, S.; Udagawa, N.; Nagata, N.; Martin, T.; Findlay, D.
1996Calcitonin Receptor Down-Regulation Relates to Calcitonin Resistance in Mature Mouse OsteoclastsWada, S.; Udagawa, N.; Nagata, N.; Martin, T.; Findlay, D.
1996Effects of Continuous Calcitonin Treatment on Osteoclast-like Cell Development and Clacitonin Receptor Expression in Mouse Bone Marrow CulturesIkegame, M.; Rakopoulos, M.; Martin, T.; Moseley, J.; Findlay, D.
1998Casein kinase 2 phosphorylation of recombinant rat osteopontin enhances adhesion of osteoclasts but not osteoblastsKatayama, Y.; House, C.; Udagawa, N.; Kazama, J.; McFarland, R.; Martin, T.; Findlay, D.