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Results 1-10 of 18 (Search time: 0.002 seconds).
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PreviewIssue DateTitleAuthor(s)
2003Hepatocyte turnover during resolution of a transient hepadnaviral infectionSummers, J.; Jilbert, A.; Yang, W.; Aldrich, C.; Saputelli, J.; Litwin, S.; Toll, E.; Mason, W.
2005Covalently closed circular DNA is the predominant form of duck hepatitis B virus DNA that persists following transient infectionLe Mire, M.; Miller, D.; Foster, W.; Burrell, C.; Jilbert, A.
2001Kinetics of hepadnavirus loss from the liver during inhibition of viral DNA synthesisZhu, Y.; Yamamoto, T.; Cullen, J.; Saputelli, J.; Aldrich, C.; Miller, D.; Litwin, S.; Furman, P.; Jilbert, A.; Mason, W.
2007Hepatocyte turnover in transient and chronic hepadnavirus infectionsMason, W.; Litwin, S.; Xu, C.; Jilbert, A.
2005Identification and characterization of avihepadnaviruses isolated from exotic anseriformes maintained in captivityGuo, H.; Mason, W.; Aldrich, C.; Saputelli, J.; Miller, D.; Jilbert, A.; Newbold, J.
2000In situ hybridization protocols for detection of viral DNA using radioactive and nonradioactive DNA probesJilbert, A.; Darby, I.
2001Sequence comparison of an Australian duck hepatitis B virus strain with other avian hepadnavirusesTriyatni, M.; Ey, P.; Tran, T.; Le Mire, M.; Qiao, M.; Burrell, C.; Jilbert, A.
2005Clonal expansion of hepatocytes during chronic woodchuck hepatitis virus infectionMason, W.; Jilbert, A.; Summers, J.
2007The liver of woodchucks chronically infected with the woodchuck hepatitis virus contains foci of virus core antigen-negative hepatocytes with both altered and normal morphologyXu, C.; Yamamoto, T.; Zhou, T.; Aldrich, C.; Frank, K.; Cullen, J.; Jilbert, A.; Mason, W.
2006DNA vaccines expressing the duck hepatitis B virus surface proteins lead to reduced numbers of infected hepatocytes and protect ducks against the development of chronic infection in a virus dose-dependent mannerMiller, D.; Kotlarski, I.; Jilbert, A.