Please use this identifier to cite or link to this item:
https://hdl.handle.net/2440/19703
Type: | Thesis |
Title: | Insulin-like growth factors and insulin-like growth factor binding proteins in wounds / James Gray Robertson |
Author: | Robertson, James Gray |
Issue Date: | 1999 |
School/Discipline: | Dept. of Surgery |
Abstract: | Thesis aimed to determine general roles for insulin-like growth factor binding proteins (IGFBPs) in regulating insulin-like growth factor-I (IGF-I) actions in wound repair. Preliminary experiments sought to characterise alterations to IGF-I levels and IGFBP profiles that may occur during wound repair. The effects that interactions with IGFBPs may have on IGF actions in wounds were addressed. Final experiments aimed to determine whether IGFBP-3 proteolysis observed in the initial work of the thesis acted to increase IGF bioavailablity. The results are discussed. |
Dissertation Note: | Thesis (Ph.D.) -- University of Adelaide, Dept. of Surgery, 2000 |
Subject: | Insulin-like growth factor-binding proteins; Wound healing Physiology; Wounds and injuries Microbiology |
Provenance: | This electronic version is made publicly available by the University of Adelaide in accordance with its open access policy for student theses. Copyright in this thesis remains with the author. This thesis may incorporate third party material which has been used by the author pursuant to Fair Dealing exception. If you are the author of this thesis and do not wish it to be made publicly available or If you are the owner of any included third party copyright material you wish to be removed from this electronic version, please complete the take down form located at: http://www.adelaide.edu.au/legals |
Appears in Collections: | Research Theses |
Files in This Item:
File | Description | Size | Format | |
---|---|---|---|---|
01front.pdf | 347.23 kB | Adobe PDF | View/Open | |
02whole.pdf | 11.12 MB | Adobe PDF | View/Open |
Items in DSpace are protected by copyright, with all rights reserved, unless otherwise indicated.